本達隆(bentazon)除草劑是萌後葉面施用型除草劑，主要用於防除闊葉雜草，其作用的目標位置為光系統Ⅱ中粒醌(plastoquinone)的結合蛋白，即QB蛋白質。此蛋白質上可供粒醌結合的位置，一旦被本達隆佔據，則粒醌無法結合於其上，將導致電子傳遞中斷而抑制光合作用，造成植物死亡。禾本科植物玉米之抗性自交系對於本達隆具抗性之原因，包括經由單加氧酶將本達隆代謝為不具毒性之葡萄糖結合物及減少除草劑的吸收、轉運等、根據遺傳研究，可知此感性基因，由單一隱性基因控制，唯基因產物之功能仍待進一步研究。

Bentazon is the herbicide effectively applied in weed control of broad-leaf weeds, which is used by foliar application postemergently. The primary action of bentazon is to compete with plastoquinone for D1 protein, results in blocking of electron transfer of PSII.Reviewing the related studies, it could be found that the bentazon-tolerant inbred lines of maize have tolerance due to detoxification of bentazon by a monooxyenase, as well as reduction of herbicide uptake and translocation. Base on the genetic studies, it was postulated that bentazon sensitivity is controlled by a single reccessive gene.